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FOX04-DRI (10mg Vial) Dosage Protocol

Table of Contents

FOXO4-DRI-10mg-main

Quickstart Highlights

FOXO4-DRI (also known as Proxofim) is a synthetic D-retro-inverso peptide designed to selectively induce apoptosis in senescent cells by disrupting the FOXO4–p53 protein interaction[1][2]. When this interaction is blocked, p53 translocates to the mitochondria in senescent cells, triggering their programmed death while sparing healthy cells[1]. This educational protocol presents a once-daily subcutaneous approach using a practical dilution for precise insulin-syringe measurements.

Dosing & Reconstitution Guide

Educational guide for reconstitution and dosing protocol

Standard / Gradual Approach (3 mL = ~3.33 mg/mL)

Week Daily Dose (mcg) Units (per injection) (mL)
Weeks 1–4 250 mcg 7.5 units (0.075 mL)
Weeks 5–8 375 mcg 11 units (0.11 mL)
Weeks 9–12 500 mcg 15 units (0.15 mL)
Weeks 13–16 500 mcg 15 units (0.15 mL)

Frequency: Inject once daily subcutaneously[7]. This schedule uses the standard 3.0 mL dilution to maintain injection volumes under 0.2 mL for all doses. For ≤10-unit (≤0.10 mL) administrations, consider 30- or 50-unit insulin syringes for improved readability.

Reconstitution Steps

Important: This guide is for educational purposes only and is not medical advice. For research use only. Not for human consumption.

Protocol Overview

Concise summary of the once-daily regimen.

Dosing Protocol

Suggested daily titration approach.

Storage Instructions

Proper storage preserves peptide quality.

Suppilies Needed

Plan based on an 8–16 week daily protocol with gradual titration.

Important Notes

Practical considerations for consistency and safety.

How This Works

FOXO4-DRI is a synthetic peptide that selectively targets senescent cells by blocking the FOXO4–p53 protein interaction[1][2]. In normal cells, FOXO4 binds to p53 and sequesters it in the nucleus. In senescent cells, disrupting this interaction causes p53 to translocate to the mitochondria, triggering apoptotic cell death specifically in aged, dysfunctional cells while sparing healthy ones[1]. Animal studies have demonstrated that FOXO4-DRI can reduce senescent cell burden, improve tissue function, and mitigate age-related pathologies[3][4][5]. The peptide’s D-retro-inverso configuration enhances its stability and resistance to proteolytic degradation[2].

 

 

Potential Benefits & Side Effects

Observations from preclinical literature.

Lifestyle Factors

Complementary strategies for optimal outcomes.

Injection Technique

General subcutaneous guidance from clinical best-practice resources[9][10][11].

Recommended Source

 We recommend Go Alpha Labs for high-purity FOXO4-DRI (10 mg).

 
 

Why Go Alpha Labs?​

Important Note:

This content is intended for therapeutic educational purposes only and does not constitute medical advice, diagnosis, or treatment.

References:

 

Source Link
Nature Communications Biology – FOXO4-DRI induces keloid senescent fibroblast apoptosis View Source
Nature Cell – Original FOXO4-DRI senescent-cell apoptosis discovery View Source
Aging (Albany NY) – FOXO4-DRI restores testosterone secretion in aged mice View Source
Journal of Cellular and Molecular Medicine – FOXO4 peptide reduces bleomycin-induced pulmonary fibrosis View Source
Cell – Senolytic mechanism context: clearance of senescent cells rejuvenates stem cells View Source
Aging (Albany NY) — Methods & Protocols – FOXO4-DRI safety note (no human clinical dosing) View Source
PubMed – Peptide metabolism assay for subcutaneous injections View Source
Bachem – Care & handling of peptides (storage, reconstitution, stability) View Source
MedlinePlus – Subcutaneous injection technique & safety View Source
American Diabetes Association – Injection site rotation guidance View Source
CDC – SQ injection angle & site (no aspiration) View Source
CDC (SQ Injection PDF) – Diagram & administration guidance View Source
NCBI Bookshelf – Best practices for injection preparation & asepsis View Source
PMC – Subcutaneous drug injection review View Source
go alpha labs – FOXO4-DRI product page (quality documentation) View Source
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